Potassium Dosing
Potassium is essential for life, growth, development & life span.
Not enough attention is paid to the potassium in our diets or in our supplements. Your daily dietary need for potassium is 2000-4000 mg. A typical modern diet only supplies 1500-5000 mg of potassium. Low potassium diets produce great fatigue & muscle weakness.
Potassium Supplements
Brand Names
Cena-K; Gen-K; K+8; K+10; Kaochlor; Kaochlor-Eff; Kaon-Cl; Kato; Kay Ciel; Kaylixir; K+Care; ET; K-Dur; K-Electrolyte; K-G Elixir; K-Gen; K-Lease; K-Lor; Klor-Con 10; Klor-Con 20; Klor-Con/25; Klorvess; Klorvess; Klyte/Cl; Klotrix; K-lyte; K-lyte; K-lyte DS; K-norm; Kolyum; k-tab; micro-K, Micro-k LS; Potasalan; rum-k; S-F Kaon; Slow-K; Ten-K; Tri-K; Trikates; Twin-K.
Potassium Citrate
Citrates (SIH-trayts) are used to make the urine more alkaline (less acid). This helps prevent certain kinds of kidney stones. Citrates are sometimes used with other medicines to help treat kidney stones that may occur with gout. They are also used to make the blood more alkaline in certain conditions. Because the citrate does make the blood and urine more alkaline this would be a poor form of Potassium for Bartter and Gitelman Patients since they do suffer with chronic metabolic alkalosis.
Support Group Rules
Like most groups, we found the need for a few rules. 
Potassium
The major intracellular ion
Potassium (K), a cation, is the most abundant cation in the body cells. Ninety-seven percent of the body’s potassium is found in the intracellular fluid (ICF) and 2-3% is found in the extracellular fluid (ECF), Which comprises of intravascular (in vessels) and interstitial fluids (between tissues). Potassium is also plentiful in the gastrointestinal tract. It is the 2-3% in the ECF that is all important in neuromuscular function. Potassium is constantly moving in and out of cells according to the body’s needs, under the influence of the sodium-potassium pump.
Magnesium Supplementation
By Shawna Kopchu RN and Herbert C. Mansmann Jr, M.D.
The recommended daily allowance for Magnesium is 6mg/kg/day. Most people need at least 100mg or as much as 600mg per day of supplemental magnesium, just to remain in Positive Magnesium Balance. Which means, one takes in more magnesium than one loses. This amount does not include the various diseases and medications that can cause you to lose Mg. If you are on any of the medications or have a disease that causes Urinary Mg Wasting then you need more than the RDA for Mg. To check the list Click Here (Need Link)
Gitelman Syndrome Information for Medical Personnel
Gitelman’s Syndrome was discovered in 1966 by Dr Hillel Gitelman. It was discovered that some patients with Bartter’s showed a different myriad of symptoms. Gitelman’s syndrome is also a renal salt wasting disorder but the defective tubule is in the thiazide-sensitive Na-Cl cotransporter in the distal convoluted tubule(DCT). Both disorders are associated with hypokalemia, renal potassium wasting, activation of the renin-angiotensin-aldosterone axis, and normal blood pressure. Unlike patients with Bartters, patients with Gitelman’s syndrome have hypomagnesemia, increased urinary magnesium, and decreased calcium excretion.
Antenatal Bartter Syndrome, Information for Medical Personnel
In contrast to Classic Bartter Syndrome and Gitelman Syndrome, the Antenatal variant of Bartter Syndrome has both the features of renal tubular hypokalemic alkalosis as well as profound systemic manifestations. Antenatal Bartter Syndrome is characterized by polyhydraminos due to intrauterine polyuria, and premature delivery is common. After birth, life-threatening episodes of fever and dehydration occur secondary to profound polyuria, vomiting, and diarrhea.
New Treatment Options for Bartter’s Syndrome
The New England Journal of Medicine
August 31, 2000 — Vol. 343, No. 9
Letter To the Editor:
Bartter’s syndrome is a major cause of congenital salt wasting. As a consequence of abnormal salt reabsorption in the thick ascending limb of Henle’s loop due to mutations in the luminal sodium-potassium-2-chloride cotransporter (antenatal Bartter’s syndrome type I), the luminal potassium channel (antenatal Bartter’s syndrome type II), or the basolateral chloride channel (classic Bartter’s syndrome type III), the activity of the renin-angiotensin-aldosterone system increases. (1) Prostaglandins increase as a consequence of volume contraction, and this increase may itself stimulate renin secretion. (2) Unfortunately, prostaglandins block salt reabsorption by mechanisms that are not yet fully understood. (1)